Lupus is characterized by attacks on the joints, skin, kidneys and immune system of the body. A fresh study demonstrates that abnormal bacteria in the intestinal tract have a good chance to increase the risk of lupus.

According to a new study conducted by scientists of Secondary schools of medicine of the new York Institute, systemic lupus erythematosus (SLE) is associated with abnormal consistency of microbes in the intestinal tract.

The disease is characterized by an attack of the body's immune system on the joints, skin and kidneys.

The creators of the true study talk, in fact, that their experiments are considered the first thorough confirmation of the relationship between bacterial imbalance in the intestinal tract and potentially life-threatening forms of SLE.

A recent study, now published in Annals of Rheumatic Diseases online, demonstrated that 61 women diagnosed with SLE had approximately 5 of the more digestive microbes, popular as Ruminococcus gnavus, than 17 ladies of similar age and racial origin, who had no disease and were healthy. Lupus is often faced with the ladies than the men.

The results of the study have proven, in fact, that the disease of the outbreak, which have all the chances to vary from cases of skin rash and joint pain to difficult renal dysfunction, requiring dialysis, carefully tracked the important increase in the rise of the microbes in the intestinal tract, in a row with the presence of immune proteins in the blood, called antibodies, formed to join the microbes.

Members of the study with renal outbreaks had a more elevated degree of antibodies to gnavus.

The senior Creator of the study, Greg Silverman, said:" Our study suggests the idea, in fact, that some patients have bacterial imbalance has the ability to be behind the wheel of reddish lupus and disease-related outbreaks, "adding," our results still show the leakage of microbes from the intestinal tract as a likely immune system Prerequisite of the disease, and hope, in fact that the internal intuition of the environment, as a consequence, have all chances to play a more significant role than genetics in renal outbreaks of this disease is very often to death. He also suspects that the antibodies to the gnavus provoke an "endless" immune attack on the organs involved in outbreaks.

Silverman added that the study has the ability to direct to the development of relatively simple blood tests to detect antibodies to leaked microbes, which, in turn, have all chances to be used to diagnose and track the progression of lupus and therapy, including at the earliest stages of the disease.

Silverman, however, warns that actually mattered more large studies, in order to prove, as these bacteria have a chance to cause lupus. But if future experiments demonstrate such flattering results, it has the ability to lead to a shift from the current scenario to the cure of disease, which focused on immunosuppressive antitumor substances for the relief of the signs and of kidney injury.